Abstract
Objective
Health-related quality of life (HRQOL) after coronary interventions (coronary artery
bypass grafting, percutaneous transluminal coronary angioplasty) usually improves
in patients, but not in all patients. Some patients actually show a significant decline
in HRQOL. Our aim was to explore the potential of psychologic well-being (anxiety,
depression), vital exhaustion, Type D personality, and socioeconomic position as predictors
of HRQOL in patients with coronary disease.
Methods
A total of 106 patients scheduled for coronary angiography were interviewed before
(baseline) and 12 to 24 months after coronary angiography. Socioeconomic status was
evaluated by education. The General Health Questionnaire 28 was used for measuring
psychologic well-being (anxiety, depression), the Maastricht interview was used for
measuring vital exhaustion, and the Type D questionnaire was used for measuring personality.
HRQOL was assessed using the Short Form-36 (physical and mental components) questionnaire.
Functional status was assessed with a combination of New York Heart Association and
Canadian Cardiovascular Society classifications. Linear regressions were used to analyze
data.
Results
A change in physical HRQOL was predicted by baseline psychologic well-being (β = −.39;
95% confidence interval [CI], −1.00 to −.16) and baseline HRQOL (β = −.61; 95% CI,
−.83 to −.34). A change in mental HRQOL was predicted by (baseline) psychologic well-being
(β = −.37; 95% CI, −.99 to −.09), vital exhaustion (β = −.21; 95% CI, −.69 to −.03),
and baseline HRQOL (β = −.76; 95% CI, −1.03 to −.44). Ejection fraction did not significantly
predict HRQOL.
Conclusion
Psychosocial factors (psychologic well-being, vital exhaustion) seem to be more important
predictors of change in HRQOL compared with some objective medical indicators (ejection
fraction) among patients with coronary disease.
Keywords
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Article info
Publication history
Published online: May 14, 2010
Accepted:
December 10,
2009
Received in revised form:
November 20,
2009
Received:
July 2,
2009
Footnotes
This work was supported by the Slovak Research and Development Agency under Contract No. APVV-20-038305 and the Project “Centre for Excellent Research of Atherosclerosis and its Complications—Myocardial Infarction and Stroke,” Operational Program of Research and Development financed by the European Fund for Regional Development.
Identification
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