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Volume 38, Issue 2, Pages 109-120 (March 2009)


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Changes in psychosocial distress in outpatients with heart failure with implantable cardioverter defibrillators

Sue A. Thomas, RN, PhD, FAANaCorresponding Author Information, Erika Friedmann, PhDa, Stephen S. Gottlieb, MD, FACCb, Fang Liu, MSa, Patricia G. Morton, RN, PhD, ACNP, FAANa, Deborah W. Chapa, RN, PhD, CRNPc, Hyeon-Joo Lee, RN, MS, ANPa, Eun-Shim Nahm, RN, PhDa, Sudden Cardiac Death in Heart Failure Trial Investigators.1

published online 29 August 2008.

Patients given implantable cardioverter defibrillators (ICDs) after arrhythmic events or sudden cardiac arrest (SCA) experience psychosocial distress. ICDs now are inserted for the primary prevention of SCA in patients with heart failure; the psychosocial impact of ICDs on patients with heart failure is unknown. Changes in psychosocial status in these ICD recipients were examined. ICD recipients (n = 57) completed depression, anxiety, and social support inventories every 6 months for up to 2 years. Initially, 35% of recipients were depressed and 45% of recipients were anxious. In linear mixed models, depression decreased over time overall but increased in those who experienced ICD shocks. Anxiety decreased in New York Heart Association class III ICD recipients but not in class II ICD recipients. Decreases in social support were related to age: the younger the patient the greater the decrease. A significant proportion of ICD recipients were depressed or anxious, or had diminished social support even after 2 years. Investigation of strategies to improve ICD recipients' psychosocial status is warranted.

a University of Maryland, School of Nursing, Baltimore, Maryland

b University of Maryland, School of Medicine, Baltimore, Maryland

c Florida Gulf Coast University, Fort Myers, Florida

Corresponding Author InformationCorresponding author: Sue Thomas, PhD, FN, FAAN, Professor and Assistant Dean for PhD Studies, University of Maryland School of Nursing, 655 W. Lombard Street, Baltimore, MD 21201-1579

 This research was partially supported by grants R01 NR07613 from the National Institute of Nursing Research, National Institutes of Health, and grants UO1 HL55766, UO1 HL55297, and UO1 HL55496 from the National Heart Lung and Blood Institute, National Institutes of Health, and by Medtronic, Wyeth–Ayerst Laboratories, and Knoll Pharmaceuticals.

1 The complete list of investigators has been published: See reference 11.

PII: S0147-9563(08)00100-3

doi:10.1016/j.hrtlng.2008.05.005


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